Citation: | Liu XZ,Li DW,Jiang M,et al.Changes in entero-insular axis function and its role in mice with severe burns[J].Chin J Burns Wounds,2024,40(7):625-633.DOI: 10.3760/cma.j.cn501225-20240520-00189. |
[1] |
Pérez-AranaGM,Díaz-GómezA,Camacho-RamírezA,et al.Dual effect of RYGB on the entero-insular axis: how GLP-1 is enhanced by surgical duodenal exclusion[J].Ann Anat,2023,249:152094.DOI: 10.1016/j.aanat.2023.152094.
|
[2] |
PorterC,TompkinsRG,FinnertyCC,et al.The metabolic stress response to burn trauma: current understanding and therapies[J].Lancet,2016,388(10052):1417-1426.DOI: 10.1016/S0140-6736(16)31469-6.
|
[3] |
LiZ,LiuX,ZhangK,et al.Role and mechanism of endoplasmic reticulum stress in mice pancreatic islet dysfunction after severe burns[J].J Burn Care Res,2023,44(5):1231-1240.DOI: 10.1093/jbcr/irad029.
|
[4] |
LiuX,LiuZ,LiD,et al.Mitochondria play a key role in oxidative stress-induced pancreatic islet dysfunction after severe burns[J].J Trauma Acute Care Surg,2022,92(6):1012-1019.DOI: 10.1097/TA.0000000000003490.
|
[5] |
张博涵,申传安,孙鹏超,等.严重烫伤大鼠早期胰岛素分泌功能变化及信号转导机制[J].中华烧伤杂志,2020,36(4):280-287.DOI: 10.3760/cma.j.cn501120-20190702-00289.
|
[6] |
ZhangB,SunP,ShenC,et al.Role and mechanism of PI3K/AKT/FoxO1/PDX-1 signaling pathway in functional changes of pancreatic islets in rats after severe burns[J].Life Sci,2020,258:118145.DOI: 10.1016/j.lfs.2020.118145.
|
[7] |
UngerRH,EisentrautAM.Entero-insular axis[J].Arch Intern Med,1969,123(3):261-266.
|
[8] |
Santos-HernándezM,ReimannF,GribbleFM.Cellular mechanisms of incretin hormone secretion[J].J Mol Endocrinol,2024,72(4):e230112.DOI: 10.1530/JME-23-0112.
|
[9] |
HolstJJ.The incretin system in healthy humans: the role of GIP and GLP-1[J].Metabolism,2019,96:46-55.DOI: 10.1016/j.metabol.2019.04.014.
|
[10] |
ShenCA,FaganS,FischmanAJ,et al.Effects of glucagon-like peptide 1 on glycemia control and its metabolic consequence after severe thermal injury--studies in an animal model[J].Surgery,2011,149(5):635-644.DOI: 10.1016/j.surg.2010.11.017.
|
[11] |
LiuX,XieX,LiD,et al.Sirt3-dependent regulation of mitochondrial oxidative stress and apoptosis contributes to the dysfunction of pancreatic islets after severe burns[J].Free Radic Biol Med,2023,198:59-67.DOI: 10.1016/j.freeradbiomed.2023.01.027.
|
[12] |
TuggleDW,KuhnMA,JonesSK,et al.Hyperglycemia and infections in pediatric trauma patients[J].Am Surg,2008,74(3):195-198.DOI: 10.1177/000313480807400302.
|
[13] |
GoreDC,ChinkesD,HeggersJ,et al.Association of hyperglycemia with increased mortality after severe burn injury[J].J Trauma,2001,51(3):540-544.DOI: 10.1097/00005373-200109000-00021.
|
[14] |
PidcokeHF,WanekSM,RohlederLS,et al.Glucose variability is associated with high mortality after severe burn[J].J Trauma,2009,67(5):990-995.DOI: 10.1097/TA.0b013e3181baef4b.
|
[15] |
LiS,LiD,LiY,et al.Development and validation of a nomogram for pneumonia risk in burn patients with inhalation injury: a multicenter retrospective cohort study[J].Int J Surg,2024,110(5):2902-2909.DOI: 10.1097/JS9.0000000000001190.
|
[16] |
van den BergheG,WoutersP,WeekersF,et al.Intensive insulin therapy in critically ill patients[J].N Engl J Med,2001,345(19):1359-1367.DOI: 10.1056/NEJMoa011300.
|
[17] |
CampbellJE,DruckerDJ.Pharmacology, physiology, and mechanisms of incretin hormone action[J].Cell Metab,2013,17(6):819-837.DOI: 10.1016/j.cmet.2013.04.008.
|
[18] |
D'AlessioD.Is GLP-1 a hormone: whether and when?[J].J Diabetes Investig,2016,7Suppl 1(Suppl 1):S50-55.DOI: 10.1111/jdi.12466.
|
[19] |
NauckMA,HombergerE,SiegelEG,et al.Incretin effects of increasing glucose loads in man calculated from venous insulin and C-peptide responses[J].J Clin Endocrinol Metab,1986,63(2):492-498.DOI: 10.1210/jcem-63-2-492.
|
[20] |
HolstJJ,GasbjergLS,RosenkildeMM.The role of incretins on insulin function and glucose homeostasis[J].Endocrinology,2021,162(7):bqab065.DOI: 10.1210/endocr/bqab065.
|
[21] |
SodumN,MattilaO,SharmaR,et al.Nutrient combinations sensed by L-cell receptors potentiate GLP-1 secretion[J].Int J Mol Sci,2024,25(2):1087.DOI: 10.3390/ijms25021087.
|
[22] |
MüllerTD,FinanB,BloomSR,et al.Glucagon-like peptide 1 (GLP-1)[J].Mol Metab,2019,30:72-130.DOI: 10.1016/j.molmet.2019.09.010.
|
[23] |
YingW,FuW,LeeYS,et al.The role of macrophages in obesity-associated islet inflammation and β-cell abnormalities[J].Nat Rev Endocrinol,2020,16(2):81-90.DOI: 10.1038/s41574-019-0286-3.
|
[24] |
LiuYT,HeT,LiHQ,et al.Liraglutide improves pancreatic islet β cell apoptosis in rats with type 2 diabetes mellitus by inhibiting the IKKε/NF-κB pathway[J].Eur Rev Med Pharmacol Sci,2021,25(14):4818-4828.DOI: 10.26355/eurrev_202107_26395.
|
[25] |
WeiR,CuiX,FengJ,et al.Dapagliflozin promotes beta cell regeneration by inducing pancreatic endocrine cell phenotype conversion in type 2 diabetic mice[J].Metabolism,2020,111:154324.DOI: 10.1016/j.metabol.2020.154324.
|
[26] |
NauckMA,QuastDR,WefersJ,et al.GLP-1 receptor agonists in the treatment of type 2 diabetes - state-of-the-art[J].Mol Metab,2021,46:101102.DOI: 10.1016/j.molmet.2020.101102.
|
[27] |
ReedJ,BainS,KanamarlapudiV.A review of current trends with type 2 diabetes epidemiology, aetiology, pathogenesis, treatments and future perspectives[J].Diabetes Metab Syndr Obes,2021,14:3567-3602.DOI: 10.2147/DMSO.S319895.
|
[28] |
LiD,ShangY,ShenC,et al.Effects of Exendin-4 on pancreatic islets function in treating hyperglycemia post severe scald injury in rats[J].J Trauma Acute Care Surg,2018,85(6):1072-1080.DOI: 10.1097/TA.0000000000002066.
|
[29] |
LiuM,WeissMA,ArunagiriA,et al.Biosynthesis, structure, and folding of the insulin precursor protein[J].Diabetes Obes Metab,2018,20Suppl 2(Suppl 2):S28-50.DOI: 10.1111/dom.13378.
|
[30] |
KwonDY,KimYS,AhnIS,et al.Exendin-4 potentiates insulinotropic action partly via increasing beta-cell proliferation and neogenesis and decreasing apoptosis in association with the attenuation of endoplasmic reticulum stress in islets of diabetic rats[J].J Pharmacol Sci,2009,111(4):361-371.DOI: 10.1254/jphs.09178fp.
|