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2016 Vol. 32, No. 2

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2016, 32(2): 65-66. doi: 10.3760/cma.j.issn.1009-2587.2016.02.001
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2016, 32(2): 66-66. doi: 10.3760/cma.j.issn.1009-2587.2016.02.101
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2016, 32(2): 81-81. doi: 10.3760/cma.j.issn.1009-2587.2016.02.102
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2016, 32(2): 88-88. doi: 10.3760/cma.j.issn.1009-2587.2016.02.103
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2016, 32(2): 96-96. doi: 10.3760/cma.j.issn.1009-2587.2016.02.104
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2016, 32(2): 109-111. doi: 10.3760/cma.j.issn.1009-2587.2016.02.010
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2016, 32(2): 112-116. doi: 10.3760/cma.j.issn.1009-2587.2016.02.011
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2016, 32(2): 117-118. doi: 10.3760/cma.j.issn.1009-2587.2016.02.012
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Expert Forum
Clinical significance of immunologic monitoring in septic patients
Yao Yongming, Li Xiuhua
2016, 32(2): 67-70. doi: 10.3760/cma.j.issn.1009-2587.2016.02.002
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Sepsis induced by burns, trauma, and surgical stress, remains a major cause of death of patients in ICUs. A growing number of evidence shows that sepsis may result in dysfunction of innate and adaptive immune system, including the abnormal disorder of immune response of T lymphocytes, regulatory T l...
Prevention and treatment strategy for burn wound sepsis in children
Niu Xihua, Li Xiaoliang
2016, 32(2): 71-73. doi: 10.3760/cma.j.issn.1009-2587.2016.02.003
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Wound sepsis is one of the main causes of death in patients with severe burn and trauma. The high incidence of burn wound sepsis in children is attributed to their imperfect immune system function, poor resistance against infection, and the weakened skin barrier function after burn. The key to reduc...
Emphasize the diagnosis and treatment of infective endocarditis in patients with severe burn
Huan Jingning
2016, 32(2): 74-76. doi: 10.3760/cma.j.issn.1009-2587.2016.02.004
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The incidence and mortality of infective endocarditis (IE) in patients with severe burn remain high, which are attributed to invasive procedures, bacteremia, and wound infection after burns. Clinical clues for IE in burns are usually masked by burn-related manifestations, so the diagnosis of IE may ...
Infection and Immunity
Prognostic significance of plasma gelsolin in severe burn patients with sepsis
Huang Lifeng, Yao Yongming, Dong Ning, He Lixin, Zhang Qinghong, Yu Yan, Sheng Zhiyong
2016, 32(2): 77-81. doi: 10.3760/cma.j.issn.1009-2587.2016.02.005
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Objective To investigate the changes in plasma gelsolin (pGSN) levels in severe burn patients with sepsis, and to evaluate the prognosis of patients when combined with other related clinical indexes. Methods Sixty-five severe burn patients with sepsis hospitalized from June 2013 to June 2015 conform...
Effects of exogenous carbon monoxide-releasing molecule 2 intervention in vitro on formation of human neutrophil extracellular traps stimulated by endotoxin/lipopolysaccharide and its mechanism
Song Mingming, Wang Xu, Qin Weiting, Zhuang Mingfeng, Xu Xiaohan, Zhang Yisen, Sun Bingwei
2016, 32(2): 82-88. doi: 10.3760/cma.j.issn.1009-2587.2016.02.006
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Objective To explore the effects of exogenous carbon monoxide-releasing molecule 2 (CORM-2) on formation of human neutrophil extracellular traps (NETs) stimulated by endotoxin/lipopolysaccharide (LPS) and its relevant mechanism. Methods Venous blood samples were collected from a healthy adult volunt...
Changes in the expression of three markers in T lymphocytes of peripheral blood and immunoregulatory mechanisms of burned mice with sepsis at early stage
Zhou Yuxiang, Huang Peng, Zhang Pihong, Ren Licheng, Zeng Jizhang, Zhou Jie, Liang Pengfei, Huang Xiaoyuan
2016, 32(2): 89-96. doi: 10.3760/cma.j.issn.1009-2587.2016.02.007
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Objective To study the expression levels of annexin A1 (ANXA1), GATA-3, and T-bet in T lymphocytes of peripheral blood in burned mice with sepsis at early stage, and to analyze their immune regulatory mechanisms. Methods Seven-hundred and eighty male mice of clean grade were divided into sham injury...
Effects of microRNA-146a on Fas-associated factor 2 and inflammatory factors in human lung adenocarcinoma A549 cells under the stimulation of cigarette smoke extract
Li Wenting, Liu Zhen, Jia Chiyu, Yin Bin, Shu Bin
2016, 32(2): 97-104. doi: 10.3760/cma.j.issn.1009-2587.2016.02.008
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Objective Under the premise of smoke inhalation injury, to explore the effects of microRNA-146a on Fas-associated factor 2 (FAF-2) and inflammatory factors in human lung adenocarcinoma A549 cells under the stimulation of cigarette smoke extract (CSE). Methods (1) The pMIR-FAF-2 recombinant plasmid a...
Original Article
Predictive value of combining of anatomy scoring system and physiological scoring system for the diagnosis of multiple organ dysfunction syndrome in patients with severe trauma
Ma Xiaoyuan, Xiao Ya, Chen Tao, Jiang Dongpo, Zhou Jian, Yan Jun, Liang Huaping
2016, 32(2): 105-108. doi: 10.3760/cma.j.issn.1009-2587.2016.02.009
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Objective To evaluate the predictive value of a combination of anatomy scoring system and physiological scoring system for the diagnosis of multiple organ dysfunction syndrome (MODS) in patients with severe trauma. Methods The clinical data of 539 patients with severe trauma hospitalized in ICUs of ...
Review
Advances in the research of mechanism of protein ubiquitination in wound healing
Wu Shan, Yao Min
2016, 32(2): 119-121. doi: 10.3760/cma.j.issn.1009-2587.2016.02.013
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Ubiquitin, a critical small molecular protein, plays an important role in regulating multiple signaling pathways. Ubiquitination is a post-translational modification induced by ubiquitin through an ATP-dependent enzyme catalyzed reaction. A large number of proteins in the complicated signaling netwo...
Advances in the research of pathogenesis and treatment of severe smoke inhalation injury
Feng Shengjuan, Jia Chiyu, Liu Zhen, Lyu Xiaowu
2016, 32(2): 122-125. doi: 10.3760/cma.j.issn.1009-2587.2016.02.014
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Among the fire victims, respiratory tract injury resulted from smoke inhalation is the major cause of death. Particulate substances in smoke, toxic and harmful gas, and chemical substances act together would rapidly induce the occurrence of dramatic pathophysiologic reaction in the respiratory tract...
Structure, function and reconstruction of skin surface lipid film
Lu Benrong, Liu Yi, Li Shilong, Wang Gang
2016, 32(2): 126-128. doi: 10.3760/cma.j.issn.1009-2587.2016.02.015
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After healing of burn wound, skin of scar, transplanted skin grafts, and healed donor site wound suffer from temporary or permanent loss of function of sebaceous glands and dysfunction of skin surface lipid film formation, resulting in desiccation, desquamation, and sensitiveness of the skin, making...

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