Citation: | Luo P,Zhang BH,Sun JC,et al.Study on the mechanism of early pancreatic exocrine function changes in severely scalded rats[J].Chin J Burns Wounds,2023,39(5):424-433.DOI: 10.3760/cma.j.cn501225-20230216-00051. |
[1] |
Dominguez-MuñozJE. Diagnosis and treatment of pancreatic exocrine insufficiency[J]. Curr Opin Gastroenterol, 2018,34(5):349-354. DOI: 10.1097/MOG.0000000000000459.
|
[2] |
ISBI Practice Guidelines Committee, SubcommitteeSteering, SubcommitteeAdvisory. ISBI practice guidelines for burn care[J]. Burns, 2016, 42(5):953-1021. DOI: 10.1016/j.burns.2016.05.013.
|
[3] |
ISBI Practice Guidelines Committee, SubcommitteeAdvisory, SubcommitteeSteering. ISBI practice guidelines for burn care, part 2[J]. Burns, 2018,44(7):1617-1706. DOI: 10.1016/j.burns.2018.09.012.
|
[4] |
PuH, DoigGS, HeighesPT, et al. Early enteral nutrition reduces mortality and improves other key outcomes in patients with major burn injury: a meta-analysis of randomized controlled trials[J]. Crit Care Med, 2018,46(12):2036-2042. DOI: 10.1097/CCM.0000000000003445.
|
[5] |
DurnoC, CoreyM, ZielenskiJ, et al. Genotype and phenotype correlations in patients with cystic fibrosis and pancreatitis[J]. Gastroenterology, 2002,123(6):1857-1864. DOI: 10.1053/gast.2002.37042.
|
[6] |
ZhangB, SunP, ShenC, et al. Role and mechanism of PI3K/AKT/FoxO1/PDX-1 signaling pathway in functional changes of pancreatic islets in rats after severe burns[J]. Life Sci, 2020, 258(118145):1-9. DOI: 10.1016/j.lfs.2020.118145.
|
[7] |
RomacJM, ShahidRA, SwainSM, et al. Piezo1 is a mechanically activated ion channel and mediates pressure induced pancreatitis[J]. Nat Commun, 2018,9(1):1715. DOI: 10.1038/s41467-018-04194-9.
|
[8] |
MeiQ, DengG, HuangZ, et al. Porous COS@SiO2 nanocomposites ameliorate severe acute pancreatitis and associated lung injury by regulating the Nrf2 signaling pathway in mice[J]. Front Chem, 2020, 8(720):1-12. DOI: 10.3389/fchem.2020.00720.
|
[9] |
DolaiS, LiangT, OrabiAI, et al. Pancreatitis-induced depletion of syntaxin 2 promotes autophagy and increases basolateral exocytosis[J]. Gastroenterology, 2018,154(6):1805-1821.e5. DOI: 10.1053/j.gastro.2018.01.025.
|
[10] |
MajumderS, ChariST. Chronic pancreatitis[J]. Lancet, 2016, 387(10031): 1957-1966. DOI: 10.1016/S0140-6736(16)00097-0.
|
[11] |
HollemansRA, HallenslebenN, MagerDJ, et al. Pancreatic exocrine insufficiency following acute pancreatitis: systematic review and study level meta-analysis[J]. Pancreatology, 2018, 18(3): 253-262. DOI: 10.1016/j.pan.2018.02.009.
|
[12] |
YangX, YaoL, FuX, et al. Experimental acute pancreatitis models: history, current status, and role in translational research[J]. Front Physiol, 2020,11:614591. DOI: 10.3389/fphys.2020.614591.
|
[13] |
JeschkeMG, van BaarME, ChoudhryMA, et al. Burn injury[J]. Nat Rev Dis Primers, 2020,6(1):11. DOI: 10.1038/s41572-020-0145-5.
|
[14] |
BittnerEA, ShankE, WoodsonL, et al. Acute and perioperative care of the burn-injured patient[J]. Anesthesiology, 2015,122(2):448-464. DOI: 10.1097/ALN.0000000000000559.
|
[15] |
AgrawalA, AlagusundarmoorthySS, JasdanwalaS. Pancreatic involvement in critically ill patients[J/OL]. J Pancreas, 2015, 16(4):346-355[2023-02-16].https://www.researchgate.net/publication/281210070_Pancreatic_Involvement_in_Critically_ill_Patients. https://www.researchgate.net/publication/281210070_Pancreatic_Involvement_in_Critically_ill_Patients
|
[16] |
SalujaA, DudejaV, DawraR, et al. Early intra-acinar events in pathogenesis of pancreatitis[J]. Gastroenterology, 2019,156(7):1979-1993. DOI: 10.1053/j.gastro.2019.01.268.
|
[17] |
ArvanitakisM, OckengaJ, BezmarevicM, et al. ESPEN guideline on clinical nutrition in acute and chronic pancreatitis[J]. Clin Nutr, 2020, 39(3):612-631. DOI: 10.1016/j.clnu.2020.01.004.
|
[18] |
HirotaM, OhmurayaM, HashimotoD, et al. Roles of autophagy and pancreatic secretory trypsin inhibitor in trypsinogen activation in acute pancreatitis[J]. Pancreas, 2020,49(4):493-497. DOI: 10.1097/MPA.0000000000001519.
|
[19] |
WilsonMD, SugdenP, DurraniA, et al. Acute pancreatitis complicating 50% full-thickness burns in a 5-year-old child[J]. Burns, 2003, 29(6):619-621. DOI: 10.1016/s0305-4179(03)00144-x.
|
[20] |
OsukaA, SugenoyaS, OnishiS, et al. Acute pancreatitis and necrotizing colitis following extensive burn injury[J]. Acute Med Surg, 2016,3(3):283-285. DOI: 10.1002/ams2.181.
|
[21] |
NeuhöferP, RoakeCM, KimSJ, et al. Acinar cell clonal expansion in pancreas homeostasis and carcinogenesis[J]. Nature, 2021,597(7878):715-719. DOI: 10.1038/s41586-021-03916-2.
|
[22] |
ApteMV, HaberPS, ApplegateTL, et al. Periacinar stellate shaped cells in rat pancreas: identification, isolation, and culture[J]. Gut, 1998,43(1):128-133. DOI: 10.1136/gut.43.1.128.
|
[23] |
CriscimannaA, SpeicherJA, HoushmandG, et al. Duct cells contribute to regeneration of endocrine and acinar cells following pancreatic damage in adult mice[J]. Gastroenterology, 2011,141(4):1451-1462, 1462.e1-e6. DOI: 10.1053/j.gastro.2011.07.003.
|
[24] |
ZhouQ, MeltonDA. Pancreas regeneration[J]. Nature, 2018,557(7705):351-358. DOI: 10.1038/s41586-018-0088-0.
|
[25] |
JiZ, LuM, XieH, et al. β cell regeneration and novel strategies for treatment of diabetes (review)[J]. Biomed Rep, 2022,17(3):72. DOI: 10.3892/br.2022.1555.
|
[26] |
GaoL, LuGT, LuYY, et al. Diabetes aggravates acute pancreatitis possibly via activation of NLRP3 inflammasome in db/db mice[J]. Am J Transl Res, 2018, 10(7):2015-2025.
|
[27] |
TybergA, KariaK, GabrM, et al. Management of pancreatic fluid collections: a comprehensive review of the literature[J]. World J Gastroenterol, 2016,22(7):2256-2270. DOI: 10.3748/wjg.v22.i7.2256.
|
[28] |
WangCC, GrossmanMI. Physiological determination of release of secretin and pancreozymin from intestine of dogs with transplanted pancreas[J]. Am J Physiol, 1951,164(2):527-545. DOI: 10.1152/ajplegacy.1951.164.2.527.
|
[29] |
MirzaKB, AlendaA, EftekharA, et al. Influence of Cholecystokinin-8 on compound nerve action potentials from ventral gastric vagus in rats[J]. Int J Neural Syst, 2018, 28(9):1850006. DOI: 10.1142/S0129065718500065.
|
[30] |
TandayN, EnglishA, LaffertyRA, et al. Benefits of sustained upregulated unimolecular GLP-1 and CCK receptor signalling in obesity-diabetes[J]. Front Endocrinol (Lausanne), 2021, 12(674704):1-13. DOI: 10.3389/fendo.2021.674704.
|