Abstract: Surgical incisions often heal with linear scars, which may further develop into hypertrophic scars or keloids. Research indicates that biomechanical factors, particularly mechanical forces and matrix stiffness, regulate scar formation by influencing cellular behavior and extracellular matrix remodeling. Our team proposes that the dynamic coupling and synergistic interactions among mechanical, chemical, and biological factors jointly regulate scar development. We emphasize that sustained tension control following wound closure is crucial to prevent scar widening and hyperplasia. Currently used tension-reducing methods have limitations: the effect of intraoperative sutures is short-lived; external tension-reducing devices (e.g., tension-reducing tape or zipper) are prone to falling off and causing skin irritation, and patient compliance is low; fractional laser may still cause the scar to widen when used alone in high-tension areas. Therefore, our team proposes a scheme utilizing an intradermal suturing technique based on slow-absorbing sutures with in-situ backstitch, which aims to achieve long-term and effective tension management. Preliminary results from clinical applications showed that this scheme achieved continuous tension reduction in surgical incisions, and its combination with fractional laser therapy is expected to synergistically improve scar width, overcoming the shortcomings of conventional methods. Moving forward, further research will optimize this scheme and promote its standardization and widespread adoption, and gradually establish a scar prevention system centered on continuous tension reduction, ultimately promoting the ideal healing of surgical incisions.